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This can be a 3-in-1 reference ebook. It supplies a whole scientific dictionary masking enormous quantities of phrases and expressions in relation to acute myelogenous leukemia. It additionally provides vast lists of bibliographic citations. eventually, it presents details to clients on how you can replace their wisdom utilizing numerous net assets. The booklet is designed for physicians, clinical scholars getting ready for Board examinations, clinical researchers, and sufferers who are looking to get to grips with study devoted to acute myelogenous leukemia. in the event that your time is effective, this ebook is for you. First, you won't waste time looking out the web whereas lacking loads of proper details. moment, the ebook additionally saves you time indexing and defining entries. eventually, you won't waste time and cash printing hundreds and hundreds of web content.
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Extra resources for Acute Myelogenous Leukemia - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References
D) PML recruits histone deacetylase and silences transcription of target genes by deacetylation of the promoter. We propose to continue to pursue the following specific aims: (1) to study the mechanism of PML regulation of cell cycle progression. Hypothesis to be tested: PML regulates cell cycle progression by inducing a GI arrest through its effects on transactivation of genes involved in the G1/S checkpoint. (2) To study the molecular basis of PML induced apoptosis. Hypothesis to be tested: PML Studies 27 induces apoptosis by (i) de-repression of the antiapoptotic effects of NFKB; (ii) induced expression of p53; and (iii) functional interaction with Bax.
The specific aims of this project are: 1. To determine the safety and biologic activity of vaccination with MAIAP in patients with AML. 2. To identify novel AML antigens through antibodybased expression cloning strategies. 3. To determine the toxicity and biologic activity of novel flt3 inhibitors in patients with AML. ; Associate Professor; Mayo Clinic Coll of Medicine, Rochester 200 1St St Sw Rochester, Mn 55905 Timing: Fiscal Year 2002; Project Start 01-APR-1998; Project End 31-JAN-2003 Summary: (adapted from the investigator's abstract) The t(7;11) in patients with acute myeloid leukemia (AML) generates a fusion gene encoding the amino-terminal NUP98, an FG repeat-containing nuclear pore complex protein (NPC), and the carboxy-terminus of HOXA9, a homeotic transcription factor.
To this end we have formed a multidisciplinary team to use human CpG island microarrays (CGI arrays) as a tool for determining CpG island methylation profiles in cancer, and from these profiles identify characteristic patterns of CpG island methylation that correlate with the tumor's clinical phenotype. The 4 integrated specific aims that follow are designed to reach our objective. 1) Construct CpG island microarrays for use in CpG island methylation analysis. 2) Optimize methylation analysis using CpG island microarrays 3) Determine CpG island methylation signatures in AML cell lines and in AML samples obtained from patients with known clinical outcome.